Supplements I Take—and Why

My supplement decisions reflect my lab results, my physicians’ guidance, and my own research. The evidence varies considerably. Some choices address a documented deficiency; others rest more on biological rationale than on proof that they prevent heart attacks.

CoQ10

My cardiologist recommended CoQ10 when he prescribed rosuvastatin, because statins can lower circulating CoQ10 levels. I take the ubiquinone form. That recommendation was my starting point—not an assumption that CoQ10 would reverse plaque or prevent a heart attack.

Vitamin D3

This one has a straightforward reason: documented deficiency. Two physicals flagged low vitamin D before I finally took it seriously. I take D3 to address that deficiency, rather than because I expect it to prevent cardiovascular events.

Vitamin K2

Given my substantial coronary calcification, I became interested in vitamin K’s role in activating proteins involved in calcium regulation. That provides a rationale for my choice, but I don’t treat K2 as a proven way to remove calcium from my arteries.

Magnesium

I include magnesium alongside D3 and K2 because of its role in normal mineral metabolism. It is part of my broader supplement regimen, rather than a treatment I can credit with changing my coronary disease.

TMG (Trimethylglycine)

I take TMG because my homocysteine has trended higher than I’d like. My goal is to address that biomarker while recognizing that lowering a lab value and proving a reduction in heart attacks are different things.

How I Evaluate Supplements

I ask the same questions I ask about the rest of my care: Why am I taking this? What evidence supports it? What can I measure? Does it still make sense alongside my medications?

This is a selection from my regimen, not a complete inventory or a recommended protocol. Supplements are an addition to my medical treatment, and I revisit my choices as my results and the evidence change.